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Recessive form caused by mutations in amphiphysin 2: bin1 homozygosity for missense mutations in bin1 was recently found in five patients from three consanguineous families to cause recessive mtm/cnm onset was congenital or in childhood.
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Affected individuals may also have an underlying predisposition to excessive activation of the na./k.-atpase pump by thyroid hormone or other factors an association with certain polymorphisms in the.-adrenergic receptor gene has been found in chinese patients, as well as an association with certain hla haplotypes treatment of the acute attacks requires cautious potassium replacement intravenously or orally, with careful monitoring of the serum k.
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